EpIdEmIology and aEtIology of cS
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چکیده
cases of endogenous CS are related to excessive adrenocorticotrophin (ACTH) production, most commonly from a pituitary adenoma, or from a non-pituitary tumour (ACTH-dependent CS). Less often endogenous CS is secondary to hyperfunctioning adrenocortical tumours, bilateral adrenal hyperplasia, and/or dysplasia (ACTH-independent CS). Endogenous CS needs to be distinguished from the so-called ‘pseudo-Cushing’s’ states, that are associated with biochemical evidence of excessive cortisol secretion without the clinical manifestations of the syndrome.
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